
自引率: 暂无数据
被引量: 2904
通过率: 暂无数据
审稿周期: 1.5
版面费用: 2500
国人发稿量: 35
投稿须知/期刊简介:
Cellular and Molecular Gastroenterology and Hepatology (CMGH) publishes cutting-edge digestive biology research that ranges from mechanisms of normal function to pathobiology and covers a broad spectrum of themes in gastroenterology, hepatology, and pancreatology. The journal reports the latest advances in cell biology, immunology, physiology, microbiology, genetics, and neurobiology of gastrointestinal, hepatobiliary, and pancreatic health and disease. CMGH publishes original papers, brief reports, reviews, editorials, commentaries, and letters to the editor. Manuscripts must be prepared in accordance with the "Uniform Requirements for Manuscripts Submitted to Biomedical Journals" developed by the International Committee of Medical Journal Editors (http://www.icmje.org). CMGH is a member of the Committee on Publication Ethics (COPE) (http://www.publicationethics.org.uk). CMGH is primarily interested in rigorous, hypothesis-driven, mechanistically novel studies that provide new insight into all aspects of gastrointestinal, hepatobiliary, and pancreatic biology. There is no requirement for work to have a disease or translational link, but studies with such implications are welcome. Most of the original research published in CMGH will take advantage of cell lines, animal models, or tissues or cells from patients. All work must be appropriately designed and powered. A founding principle of CMGH is that the wide dissemination of outstanding research is critical to progress in digestive health and disease. Therefore, CMGH is a completely digital, open access journal, meaning that its content is easily and freely accessible from around the globe.
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Sphingosine Kinase 1 Aggravates Liver Fibrosis by Mediating Macrophage Recruitment and Polarization.
被引量:- 发表:1970
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RBM39 Enhances Cholangiocarcinoma Growth Through EZH2-mediated WNT7B/β-catenin Pathway.
被引量:- 发表:1970
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Intestinal Stearoyl-CoA Desaturase-1 Regulates Energy Balance via Alterations in Bile Acid Homeostasis.
被引量:- 发表:1970
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The Progress and Promise of Lineage Reprogramming Strategies for Liver Regeneration.
The liver exhibits remarkable regenerative capacity. However, the limited ability of primary human hepatocytes to proliferate in vitro, combined with a compromised regenerative capacity induced by pathological conditions in vivo, presents significant obstacles to effective liver regeneration following liver injuries and diseases. Developing strategies to compensate for the loss of endogenous hepatocytes is crucial for overcoming these challenges, and this remains an active area of investigation. Lineage reprogramming, the process of directly converting one cell type into another bypassing the intermediate pluripotent state, has emerged as a promising method for generating specific cell types for therapeutic purposes in regenerative medicine. Here, we discuss the recent progress and emergent technologies in lineage reprogramming into hepatic cells, and their potential applications in enhancing liver regeneration or treating liver disease models. We also address controversies and challenges that confront this field.
被引量:- 发表:1970
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BET Inhibition Rescues Acinar-Ductal-Metaplasia and Ciliogenesis and Ameliorates Chronic Pancreatitis-Driven Changes in Mice With Loss of the Polarity Protein Par3.
被引量:- 发表:1970