自引率: 5.7%
被引量: 1770
通过率: 暂无数据
审稿周期: 2
版面费用: 暂无数据
国人发稿量: 17
投稿须知/期刊简介:
Published by Zhejiang University School of Medicine, China. ISSN: 1499-3872.<br /><br />Hepatobiliary & Pancreatic Diseases International (HBPD INT) publishes peer-reviewed original papers, reviews and editorials concerned with clinical practice and research in the fields of hepatobiliary and pancreatic diseases. Papers cover the medical, surgical, radiological, pathological, biochemical, physiological and historical aspects of the subject areas under the headings Liver, Biliary, Pancreas, Transplantation, Research, Special Reports, Editorials, Review Articles, Brief Communications, Clinical Summary, Clinical Images and Case Reports. The journal also deals with the basic sciences and experimental work. All submitted papers are reviewed by at least two referees expert in the field of the submitted paper. For detailed instructions concerning the submission of manuscripts, please refer to the Instructions for Authors in each issue of the journal. HBPD INT is published bimonthly since 2007.
期刊描述简介:
Published by Zhejiang University School of Medicine, China. ISSN: 1499-3872. Hepatobiliary & Pancreatic Diseases International (HBPD INT) publishes peer-reviewed original papers, reviews and editorials concerned with clinical practice and research in the fields of hepatobiliary and pancreatic diseases. Papers cover the medical, surgical, radiological, pathological, biochemical, physiological and historical aspects of the subject areas under the headings Liver, Biliary, Pancreas, Transplantation, Research, Special Reports, Editorials, Review Articles, Brief Communications, Clinical Summary, Clinical Images and Case Reports. The journal also deals with the basic sciences and experimental work. All submitted papers are reviewed by at least two referees expert in the field of the submitted paper. For detailed instructions concerning the submission of manuscripts, please refer to the Instructions for Authors in each issue of the journal. HBPD INT is published bimonthly since 2007.
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Liver transplantation for hepatocellular carcinoma: Current status in Hong Kong, China.
被引量：- 发表：1970
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Chronic viral hepatitis B complicated by hepatitis E virus infection.
被引量：- 发表：1970
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Borderline resectable giant hepatic cavernous hemangioma and coexisting hemangiomatosis should be a new indication for living donor liver transplantation: A report of two cases.
被引量：- 发表：1970
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The crosstalk between cellular survival pressures and N6-methyladenosine modification in hepatocellular carcinoma.
Within the tumor microenvironment, survival pressures are prevalent with potent drivers of tumor progression, angiogenesis, and therapeutic resistance. N6-methyladenosine (m6A) methylation has been recognized as a critical post-transcriptional mechanism regulating various aspects of mRNA metabolism. Understanding the intricate interplay between survival pressures and m6A modification provides new insights into the molecular mechanisms underlying hepatocellular carcinoma (HCC) progression and highlights the potential for targeting the survival pressures-m6A axis in HCC diagnosis and treatment. A literature search was conducted in PubMed, MEDLINE, and Web of Science for relevant articles published up to April 2024. The keywords used for the search included hepatocellular carcinoma, cellular survival, survival pressure, N6-methyladenosine, tumor microenvironment, stress response, and hypoxia. This review delves into the multifaceted roles of survival pressures and m6A RNA methylation in HCC, highlighting how survival pressures modulate the m6A landscape, the impact of m6A modification on survival pressure-responsive gene expression, and the consequent effects on HCC cell survival, proliferation, metastasis, and resistance to treatment. Furthermore, we explored the therapeutic potential of targeting this crosstalk, proposing strategies that leverage the understanding of survival pressures and m6A RNA methylation mechanisms to develop novel, and more effective treatments for HCC. The interplay between survival pressures and m6A RNA methylation emerges as a complex regulatory network that influences HCC pathogenesis and progression.
被引量：- 发表：1970
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Cirrhosis after pancreaticoduodenectomy for pancreatic cancer.
被引量：- 发表：1970
