
自引率: 3.1%
被引量: 6360
通过率: 暂无数据
审稿周期: 2
版面费用: 暂无数据
国人发稿量: 8
投稿须知/期刊简介:
Overview<br clear="none">Traffic covers the cell biology and biochemistry of intracellular transport in health and disease and aims to publish manuscripts at the forefront of this field. Specific topics are outlined in the Aims & Scope.<br clear="none">The Editors of Traffic have signed up to The San Francisco Declaration on Research Assessment (DORA). For more information please visit the DORA website.Aims and Scope<br clear="none">Traffic encourages and facilitates the publication of papers covering the cell biology of intracellular transport in health and disease. Areas of interest include protein, nucleic acid and lipid traffic, molecular motors, intracellular pathogens, intracellular proteolysis, nuclear import and export, cytokinesis and the cell cycle, protein translocation, antigen processing and presentation, organelle biogenesis, cell polarity and organization, and organelle movement.<br clear="none"><br clear="none">All aspects of the structural, molecular biology, biochemistry, genetics, morphology, intracellular signaling and relationship to hereditary or infectious diseases will be covered. Manuscripts must provide a clear conceptual or mechanistic advance. The editors will reject papers that require major changes, including addition of significant experimental data or other significant revision.<br clear="none"><br clear="none">Traffic will consider manuscripts of any length, but encourages authors to limit their papers to 16 typeset pages or lessKeywords<br clear="none">actin, adipocyte cell biology, antigen processing and presentation, cell adhesion, Cell modeling, cell morphogenesis, chloroplast assembly, chloroplast protein export, chloroplast protein import, chloroplasts, cilia, cilia assembly, ciliary function, cytokinesis, cytoskeletal function, endocytosis, endoplasmic reticulum, endosomes, ERGIC, G-protein coupled receptors, glycolipid assembly and trafficking, glycoprotein assembly and trafficking, Golgi Complex, hereditary diseases of membrane trafficking, immune cell biology, Immunity, inflammation, intracellular motility, intracellular pathogens, intracellular proteolysis, lipid bodies, lipid droplets, lipid trafficking, lysosomal storage diseases, lysosome-related organelles, lysosomes, membrane fission, membrane fusion, Membrane trafficking, microfilaments, microtubule motors, microtubules, mitochondria, mitochondrial fission and fusion, mitochondrial protein import, Model organisms, model systems, Muscle cell biology, myosins, nuclear export, nuclear import, Nuclear lamina, nuclear structure, nuclear transport, Nucleus, organelle biogenesis, organelle motility, parasite cell biology, peroxisome assembly, peroxisome protein import, phagocytosis, phagosome maturation, plastids, protein complex assembly, protein folding, protein structure, protein trafficking, protein translocation, quality control, receptor down-regulation, Regulated secretion, secretion, secretory granules, secretory packages, subcellular organelles, vacuole, vesicle, vesicular transport carriers, viral trafficking, virus assembly, virus cell biology, virus entry Abstracting and Indexing Information<br clear="none">Academic Search (EBSCO Publishing)Academic Search Alumni Edition (EBSCO Publishing)Academic Search Premier (EBSCO Publishing)AGRICOLA Database (National Agricultural Library)CSA Biological Sciences Database (ProQuest)Current Contents: Life Sciences (Thomson Reuters)Embase (Elsevier)Journal Citation Reports/Science Edition (Thomson Reuters)MEDLINE/PubMed (NLM)Neurosciences Abstracts (ProQuest)PubMed Dietary Supplement Subset (NLM)Research Alert (Thomson Reuters)Science Citation Index Expanded (Thomson Reuters)SCOPUS (Elsevier)
期刊描述简介:
Traffic covers the cell biology and biochemistry of intracellular transport in health and disease and aims to publish manuscripts at the forefront of this field. The aim of Traffic is to publish papers that provide insight into the mechanisms that underlie subcellular trafficking, membrane dynamics, and organelle function and how these subcellular processes influence cellular physiology. Specific topics are outlined in the Aims & Scope below. The Editors of Traffic have signed up to The San Francisco Declaration on Research Assessment (DORA). For more information please visit the DORA website. Traffic publishes the editorial correspondence related to the review of accepted papers as supplemental material online. This includes the decision letter(s), referee comments and author rebuttals. Referee identities and confidential comments to the editor will remain confidential.
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Post-Transcriptional Regulation of Rab7a in Lysosomal Positioning and Drug Resistance in Nutrient-Limited Cancer Cells.
被引量:1 发表:2024
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Dissociation of Drosophila Evi-Wg Complex Occurs Post Apical Internalization in the Maturing Acidic Endosomes.
被引量:- 发表:2024
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Mechanistic Insights Into an Ancient Adenovirus Precursor Protein VII Show Multiple Nuclear Import Receptor Pathways.
被引量:- 发表:2024
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Intercellular Mitochondrial Transfer: The Novel Therapeutic Mechanism for Diseases.
Mitochondria, the dynamic organelles responsible for energy production and cellular metabolism, have the metabolic function of extracting energy from nutrients and synthesizing crucial metabolites. Nevertheless, recent research unveils that intercellular mitochondrial transfer by tunneling nanotubes, tumor microtubes, gap junction intercellular communication, extracellular vesicles, endocytosis and cell fusion may regulate mitochondrial function within recipient cells, potentially contributing to disease treatment, such as nonalcoholic steatohepatitis, glioblastoma, ischemic stroke, bladder cancer and neurodegenerative diseases. This review introduces the principal approaches to intercellular mitochondrial transfer and examines its role in various diseases. Furthermore, we provide a comprehensive overview of the inhibitors and activators of intercellular mitochondrial transfer, offering a unique perspective to illustrate the relationship between intercellular mitochondrial transfer and diseases.
被引量:- 发表:2024
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Intracellular Trafficking Defects in Congenital Intestinal and Hepatic Diseases.
Enterocytes and liver cells fulfill important metabolic and barrier functions and are responsible for crucial vectorial secretive and absorptive processes. To date, genetic diseases affecting metabolic enzymes or transmembrane transporters in the intestine and the liver are better comprehended than mutations affecting intracellular trafficking. In this review, we explore the emerging knowledge on intracellular trafficking defects and their clinical manifestations in both the intestine and the liver. We provide a detailed overview including more investigated diseases such as the canonical, variant and associated forms of microvillus inclusion disease, as well as recently described pathologies, highlighting the complexity and disease relevance of several trafficking pathways. We give examples of how intracellular trafficking hubs, such as the apical recycling endosome system, the trans-Golgi network, lysosomes, or the Golgi-to-endoplasmic reticulum transport are involved in the pathomechanism and lead to disease. Ultimately, understanding these processes could spark novel therapeutic approaches, which would greatly improve the quality of life of the affected patients.
被引量:- 发表:2024