自引率: 2.9%
被引量: 34378
通过率: 暂无数据
审稿周期: 1.33
版面费用: 暂无数据
国人发稿量: 108
投稿须知/期刊简介:
The Journal of Neurochemistry is the leading source for current research worldwide on the molecular chemical and cellular biology of the nervous system. Each issue contains dozens of full-length presentations of significant original findings written by investigators at leading medical and research institutions around the world. The Journal of Neurochemistry is devoted to the prompt publication of high-quality original findings in areas relevant to molecular chemical and cell biological aspects of the nervous system. Papers that are wholly pharmalogical histochemical or immunological and methods papers or the cloning of confirmatory sequences that do not advance knowledge in neurochemistry are not normally considered. The Journal particularly encourages submissions in the areas of molecular and cellular biology. A highlight of each issue is the Journal's critically acclaimed Rapid Communications section presenting new ideas and data of particular importance and timeliness. The Journal's Mini-Reviews present concise self-contained summaries of current research in particularly important areas.
期刊描述简介:
The Journal of Neurochemistry is the leading source for current research worldwide on the molecular chemical and cellular biology of the nervous system. Each issue contains dozens of full-length presentations of significant original findings written by investigators at leading medical and research institutions around the world. The Journal of Neurochemistry is devoted to the prompt publication of high-quality original findings in areas relevant to molecular chemical and cell biological aspects of the nervous system. Papers that are wholly pharmalogical histochemical or immunological and methods papers or the cloning of confirmatory sequences that do not advance knowledge in neurochemistry are not normally considered. The Journal particularly encourages submissions in the areas of molecular and cellular biology. A highlight of each issue is the Journal's critically acclaimed Rapid Communications section presenting new ideas and data of particular importance and timeliness. The Journal's Mini-Reviews present concise self-contained summaries of current research in particularly important areas.
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The longitudinal behavioral effects of acute exposure to galactic cosmic radiation in female C57BL/6J mice: Implications for deep space missions, female crews, and potential antioxidant countermeasures.
被引量：- 发表：1970
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Chronic in vivo sequelae of repetitive acute mfb-DBS on accumbal dopamine and midbrain neuronal activity.
被引量：- 发表：1970
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Mass spectrometry identifies tau C-terminal phosphorylation cluster during neuronal hyperexcitation.
被引量：- 发表：1970
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RETRACTION: Extensive degradation of myelin basic protein isoforms by calpain following traumatic brain injury.
Retraction: M. C. Liu, V. Akle, W. Zheng, J. Kitlen, B. O'Steen, S. F. Larner, J. R. Dave, F. C. Tortella, R. L. Hayes, and K. K. W. Wang, "Extensive Degradation of Myelin Basic Protein Isoforms by Calpain Following Traumatic Brain Injury," Journal of Neurochemistry 98, no. 3 (2006): 700-712. https://doi.org/10.1111/j.1471-4159.2006.03882.x. The above article, published online on 19 June 2006, in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Andrew Lawrence; the International Society for Neurochemistry; and John Wiley and Sons Ltd. A third party reported that they had detected evidence of image manipulation and duplication throughout the published article. An investigation by the publisher and the editors concluded that there was evidence of splicing in Figures 2a, 2b, 3a, 5a, 6a, and 8a. The investigation also found duplications of lanes in Figures 3a, 3d, and 4a. Lastly, the investigation found that the same actin blots in Figure 3 have been used in two other articles by many of the same authors, despite representing different experimental conditions. The authors did not respond to an inquiry by the publisher. The retraction has been agreed to because the results presented in the article can no longer be considered reliable. The authors did not respond to the notice of retraction.
被引量：- 发表：1970
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Compensatory changes after spinal cord injury in a remyelination deficient mouse model.
被引量：1 发表：1970
